Cohort Studyhigh evidence
A terminal metabolite of niacin promotes vascular inflammation and contributes to cardiovascular disease risk.
Nature medicine·February 1, 2024·PMID 38374343
Why it mattersNiacin is widely consumed through fortified foods and supplements, yet its cardiovascular effects remain controversial. This paper identifies a potential pathway by which niacin excess could promote vascular inflammation and residual risk, offering a new target for risk stratification and therapeutic intervention.
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